Category: autism

Summarizing Some Sallis Theories

Some Sallis theories.

The Sallis solution to the Fermi Paradox:

…racial diversity is the rule among sentient species; thus, highly divergent human racial types find their counterparts in significant racial variability among the alien peoples inhabiting worlds unknown. I further propose a Gresham’s law of universal racial diversity: the more intelligent and productive races of any sentient species will tend to be demographically outcompeted by the more stupid and useless races; the latter, despite their deficiencies for proximate values, are inevitably more fit with respect to the ultimate criterion of survival.  The threshold of technological competence for a detectable alien civilization would no longer obtain as the race or races capable of maintaining that civilization are replaced by those incapable.  Therefore, no such civilizations are detected.

The Sallis theory of autism.  Thus, the race recognition processing software of the amygdala (also involved in threat recognition) of Whites, particularly White children, is capable of easily distinguishing highly distinct racial types – Europeans, sub-Saharan Africans, (pure) Amerindians, and East Asians.  But the post-1965 immigration influx into America (and similar immigration influxes into Europe) has brought into White societies previously rare, more racially intermediate types – South Asians, heavily admixed Latin Americans, Middle/Near Easterners and North Africans. This phenotypically confusing racial diversity “scrambles” the amygdala of susceptible White children, increasing risk for autism.  Hence, the link between diversity and autism and, hence, an explanation for some of the population-autism correlations discovered by James Bowery some years ago.

As regards politics – my “Sallis strategy.”

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Xenophobia Is Good Mental Health

There are many legitimate forms of trepidation.

Note that damage to the amygdala can result in an individual unable to recognize dangerous situations, an individual always in trouble, and an individual whose genetic interests are endangered by the inability to actualize prudent and reasonable caution.

The amygdala is also important for race recognition, and I have previously written about my hypothesis on autism, that ties together the various functions of this brain structure into a coherent whole, and which points put the potential dangers of South Asian phenotypes to young White children.

Getting back to the main point here: can we not say that an absence of xenophobia – in other words, typical liberal universalism – is akin to a defect in the amygdala that impairs the recognition of the danger of the racial other?  Isn’t the typical xenophile leftist akin to someone walking around with a damaged brain with a damaged amygdala?  Isn’t racial liberalism than akin to brain damage, and isn’t then xenophobia akin to mental and neurological good health?

Then we get into the question of race-mixing.

Putting aside the issue of casual miscegenation (which is bad enough), let us consider the gross enormity of actually producing children with someone of a different race; let us consider the horror of inter-racial marriage. Of inter-racial family formation: spending your life with, and reproducing with, an alien life form.  What can we see about the amygdala of such a person?  What kind of defect must hey have in their mentality, in their brain circuitry?  Can any such person be trusted?  Why would WNs embrace any individual who “sleeps with the enemy” and who donates their genetic material to produce more of your racial enemies?  Aren’t the ones who embrace such individuals mentally damaged themselves?

The Amygdala, Race, and Autism

An interesting connection.


An increased pattern of brain activity in the amygdalas of adults with autism that may be linked to the social deficits that typically are associated with the disorder. Previous research has shown that abnormal growth patterns in the amygdala are commonly found among young children diagnosed with autism. 


Replicating past research, greater amygdala activity was observed for Black faces than White faces. Furthermore, dark-skinned targets elicited more amygdala activity than light-skinned targets. However, these results were qualified by a significant interaction between race and skin tone, such that amygdala activity was observed at equivalent levels for light- and dark-skinned Black targets, but dark-skinned White targets elicited greater amygdala activity than light-skinned White targets. 


Both race and skin tone affect amygdala activity.  My original South Asian theory of White autism hypothesized that the ‘intermediate” phenotypes of South Asians – Caucasian “(“White”) but with dark skins, and with facial features typically different from that of European Whites – confuses the race recognition “software” of the amygdala in young White children, damaging the amygdala “hardware” as a result, leading to aberrant development and function of that brain structure, finally resulting in autism.

Is this the reason autism rates have increased in White countries with the rise of NEC immigration (1965 Immigration Act in America, and the North African/Middle Eastern flood in Europe)?  And, in general, overall increases in inter-continental population diversity seems associated with increased rates of autism. Maybe East Asian phenotypes are responsible as well? That would seem likely.