Category: IQ

Strom Contra Derbyshire

The superiority of WN 1.0 is shown once again.

Read this, emphasis added:

It means that the majority of racial mixing involves the destruction of the White race – Whites mating with Asians, Whites mating with Blacks, Whites mating with Arabs or Jews, Whites mating with mestizos, Whites mating with the racially unclassifiable. You have seen it in your shopping centers. You have seen it in the street. You are a witness to genocide. You are seeing it before your very eyes every day. What are you doing about it? If you do not at least speak out against it, you are allowing yourself to be complicit in this horrible crime.

The crime is racemixing. It is a worse crime than murder – far worse.

For when you commit murder you kill one man, you end one life, you tragically injure one family and circle of friends. When you commit murder, if your victim has had no children you do cut off the potential existence of one small branch of the race’s future.

But when you commit the crime of racial mixing you are participating in genocide. The probable effect and possible motive for your act is to bring into the world hybrid young, who will not be clearly of one race or the other and which will, by their very existence, increase the probability of future racial mixing and dilute both the gene pool and the sense of identity of the next generation of White children.

Derbyshire is even worse than what Strom describes here, since he not only has race-mixed, but has written for VDARE defending race-mixing and asserting that “racial purists” are mentally unstable (“slightly nuts”).

When will people like Strom denounce Derbyshire as this blog does? After all, Kevin, aren’t you being complicit to White genocide by not openly denouncing the fact that Derbyshire is being given a forum to disseminate his views on forums of the Far Right?

On another, perhaps not completely unrelated note:

This finding has been observed most strongly in Der Movement, I suppose.

Advertisements

We Are Not All the Same

Genes, Race, IQ, and disease.

One refutation of Lynn, and three papers with emphasis added.

Refuting Lynn, refuting the Alt Wrong/Alt Yellow.  Amren weeps.

Read here.

BACKGROUND:
Although cell lines are an essential resource for studying cancer biology, many are of unknown ancestral origin, and their use may not be optimal for evaluating the biology of all patient populations.
METHODS:
An admixture analysis was performed using genome-wide chip data from the Catalogue of Somatic Mutations in Cancer (COSMIC) Cell Lines Project to calculate genetic ancestry estimates for 1018 cancer cell lines. After stratifying the analyses by tissue and histology types, linear models were used to evaluate the influence of ancestry on gene expression and somatic mutation frequency.
RESULTS:
For the 701 cell lines with unreported ancestry, 215 were of East Asian origin, 30 were of African or African American origin, and 453 were of European origin. Notable imbalances were observed in ancestral representation across tissue type, with the majority of analyzed tissue types having few cell lines of African American ancestral origin, and with Hispanic and South Asian ancestry being almost entirely absent across all cell lines. In evaluating gene expression across these cell lines, expression levels of the genes neurobeachin line 1 (NBEAL1), solute carrier family 6 member 19 (SLC6A19), HEAT repeat containing 6 (HEATR6), and epithelial cell transforming 2 like (ECT2L) were associated with ancestry. Significant differences were also observed in the proportions of somatic mutation types across cell lines with varying ancestral proportions.
CONCLUSIONS:
By estimating genetic ancestry for 1018 cancer cell lines, the authors have produced a resource that cancer researchers can use to ensure that their cell lines are ancestrally representative of the populations they intend to affect. Furthermore, the novel ancestry-specific signal identified underscores the importance of ancestral awareness when studying cancer.

Racial genetic differences mean that results obtained with cancer cell lines from one race may very well be NOT applicable to other races.  There are indeed racial differences in gene sequences and gene expression, with clinically significant implications for patients.

Read here.

BACKGROUND:
We examined racial differences in the expression of eight genes and their associations with risk of recurrence among 478 white and 495 black women who participated in the Carolina Breast Cancer Study Phase 3.
METHODS:
Breast tumor samples were analyzed for PAM50 subtype and for eight genes previously found to be differentially expressed by race and associated with breast cancer survival: ACOX2, MUC1, FAM177A1, GSTT2, PSPH, PSPHL, SQLE, and TYMS. The expression of these genes according to race was assessed using linear regression and each gene was evaluated in association with recurrence using Cox regression.
RESULTS:
Compared to white women, black women had lower expression of MUC1, a suspected good prognosis gene, and higher expression of GSTT2, PSPHL, SQLE, and TYMS, suspected poor prognosis genes, after adjustment for age and PAM50 subtype. High expression (greater than median versus less than or equal to median) of FAM177A1 and PSPH was associated with a 63% increase (hazard ratio (HR) = 1.63, 95% confidence interval (CI) = 1.09-2.46) and 76% increase (HR = 1.76, 95% CI = 1.15-2.68), respectively, in risk of recurrence after adjustment for age, race, PAM50 subtype, and ROR-PT score. Log2-transformed SQLE expression was associated with a 20% increase (HR = 1.20, 95% CI = 1.03-1.41) in recurrence risk after adjustment. A continuous multi-gene score comprised of eight genes was also associated with increased risk of recurrence among all women (HR = 1.11, 95% CI = 1.04-1.19) and among white (HR = 1.14, 95% CI = 1.03-1.27) and black (HR = 1.11, 95% CI = 1.02-1.20) women.
CONCLUSIONS:
Racial differences in gene expression may contribute to the survival disparity observed between black and white women diagnosed with breast cancer.

Health disparity differences in outcome for breast cancer in White vs. Black women have a genetic basis.

Read this.

Age at menarche (AM) and age at natural menopause (ANM) define the boundaries of the reproductive lifespan in women. Their timing is associated with various diseases, including cancer and cardiovascular disease. Genome-wide association studies have identified several genetic variants associated with either AM or ANM in populations of largely European or Asian descent women. The extent to which these associations generalize to diverse populations remains unknown. Therefore, we sought to replicate previously reported AM and ANM findings and to identify novel AM and ANM variants using the Metabochip (n = 161,098 SNPs) in 4,159 and 1,860 African American women, respectively, in the Women’s Health Initiative (WHI) and Atherosclerosis Risk in Communities (ARIC) studies, as part of the Population Architecture using Genomics and Epidemiology (PAGE) Study. We replicated or generalized one previously identified variant for AM, rs1361108/CENPW, and two variants for ANM, rs897798/BRSK1 and rs769450/APOE, to our African American cohort. Overall, generalization of the majority of previously-identified variants for AM and ANM, including LIN28B and MCM8, was not observed in this African American sample. We identified three novel loci associated with ANM that reached significance after multiple testing correction (LDLR rs189596789, p = 5×10⁻⁰⁸; KCNQ1 rs79972789, p = 1.9×10⁻⁰⁷; COL4A3BP rs181686584, p = 2.9×10⁻⁰⁷). Our most significant AM association was upstream of RSF1, a gene implicated in ovarian and breast cancers (rs11604207, p = 1.6×10⁻⁰⁶). While most associations were identified in either AM or ANM, we did identify genes suggestively associated with both: PHACTR1 and ARHGAP42. The lack of generalization coupled with the potentially novel associations identified here emphasize the need for additional genetic discovery efforts for AM and ANM in diverse populations.

There seems to be genetic differences underlying reproductive lifespan in women of different races.  I hypothesize that Negro females would tend to possess variants promoting earlier reproduction.  Blacks and Hispanics have earlier puberty than Whites.

Der Movement Marches On

More wreckage.

Whatever you want to say about Taleb, he’s at least forcing the Alt Wrong HBD fetishists to defend their premises – for the first time, in, well, forever.

Apparently, those 23ndMe results have real world consequences.

A bit behind the times:

December 2018.

August 2017.

Never fear – given enough time, Der Movement belatedly realizes, promotes, or understands many of the points raised here months or years earlier.

Does the taint of Arktos prevent book reviews?

Vincere Arkadin
Posted December 26, 2018 at 5:27 am | Permalink
What about Bolton’s Yockey bio? Did you read it by chance?

Greg Johnson

Posted December 26, 2018 at 5:37 am | Permalink
I have not been able to read it.

A Type II review is here.

Meanwhile, Johnson is now a writer for the Jewish-HBD Unz site.  Zman would be proud. Is this a paid position?  After all, moneybags Ron usually pays his writers.  Does Greg believe that Unz should be “driven out of White society” as he has stated for Jews in general?  Pay close attention, goys, pay close attention.

The young Pirandello.

The young Mussolini.

More examples for the “movement” cherry pickers:

Giovanni Gentile.

In phenotypic contrast, Lega Nord politician Maroni, born in Varese, Lombardy.  Undoubtedly, a Celt-Germanic Nordic.  As Roissy commentators assert – “geography matters in Italy, like a lot.”

For Greg Conte – an Italian politician of Calabrian ancestry.  Another purebred mountain wop, no doubt. Arya…arya….

Go away, Parrott.

This is the future you choose for your children when you mumble on with your excuses and reservations.
Yes, it’s worth some people getting doxxed, fired, and arrested over. It’s a real struggle, not an internet hobby.

Rewritten:

This is the future you choose for your children when you tolerate outrageously incompetent quota queens, freakshows, and online grifters.
Yes, it’s worth some “movement leaders” being mocked and ridiculed. It’s a real struggle, not a panhandling scheme for people who want to live off of follower’s donations.

Der movement marches on.  The voices of WN 3.0?  Say it aint’ so!  Let’s see now.  Trolling, “shitposting,” and generalized juvenile jackassery: all WN 2.0 activities.  Another fail.  Is Greg going to try and blame this fiasco on WN 1.0 as well?

And this is another example why Parrott and all the other quota queens are so wrong in their “suggestions” and “advice.”  Rallies, trolling, beer bellies with their muskets, and all the rest mean nothing, absolutely nothing, when there is no infrastructure, no one embedded in the System, not a single lawyer on call ready to defend, absolutely nothing, nada, zip, zero – nothing except drunken podcasting, Pepe and Kek, trailer park cuckolding, “Proud Boys” chased out of LA boys and then abandoned by their founder, and “leaders” constantly with their hands out demanding “D’Nations.”  There’s nothing.  Absolutely nothing.  And if anyone asserts that we need something, then the good old boys emerge out of the woodwork and – aping their retarded God Emperor – lash out with tweets.  Pathetic.

We’re stuck in an eternal negative feedback loop.  The freakshows of Der Movement repel high quality people.  The lack of high quality people leads to a “movement” dominated by freaks, incompetents, and jackasses. This in turn further repels high quality people.

Only when the rank-and-file activists rebel against their “leaders” will this ever change.

Merry Christmas 2018

In all cases, emphasis added.

Read here.

And what is Christmas without Brimelow using his young children as props for some implicit tin cup panhandling?  Although here it is more implicit and more indirect than usual:

So I more than ever want to wish a Merry, and blessed, Christmas to our readers, writers and especially donors—God bless you, every one.

Donors are especially blessed; after all, you make things like this possible!  Now, that’s some moral seriousness, to be sure!  What better way to spend “movement” donations?

Notice how Asians rank here.  Closer to Blacks and Hispanics than even to Jews.  Colored is as colored does.  Asians: A more intelligent form of Negro.

I have mixed opinions about Taleb’s criticism of the worth of IQ.  On the one hand, I agree with him that some people over-rate the importance of IQ.  After all, I am one of the leading Far Right critics of HBD and the Alt Wrong, and I have criticized the HBDers’ fetishistic obsession with IQ.  There are indeed “intellectual idiots” out there – consider how many academics are dogmatic leftists, spouting the most absurd nonsense.  We also have such types on our side as well. So, yes, IQ is not a complete measure of human worth, and a high IQ does not automatically equate with success.  Note the extreme deficiencies of high-IQ Jews and Asians, as well as high-IQ Whites who cannot understand fundamental realities in the midst of their demented intellectualized fantasies. But…

On the other hand, Taleb goes too far.  Just because something is over-rated does not mean it is not vitally important.  Just because something is not sufficient does not mean it is not necessary.  The entire modern world has been built by high-IQ (European-derived male) individuals, and low-IQ areas are cesspools of filth and degradation.  The following riposte to Taleb is also cogent:

It’s funny to see someone with a genius level IQ like Taleb (a talented quant and professor) literally deny the importance of the metric, when he himself no doubt has a very high IQ, and would not have the cognitive abilities or success he enjoys without it.

It’s easy to be a contrarian and take a controversial position that focuses attention on your views; it is more practical to see the positive and negatives of different human traits.  Ironically enough, Taleb’s own criticism of IQ is precisely the type of intellectualized theorizing, independent of real world common sense, that he rails against. A pragmatic view would be that IQ is fundamentally important but incomplete; it is simply one component of a person’s mental toolkit, only one part of their cognitive and behavioral portfolio.  Taleb instead takes an impractical, rigidly dogmatic approach to the subject and he is wrong for doing so.

To put it in juvenile Alt Right language: Taleb is “sperging out” as badly as any IQ-obsessed ivory tower intellectual.

What is Rudolph’s cephalic index?  Durocher, get out your calipers!

Hey!  I thought – according to Roissy and his commentators – that Whitaker was a “Bane” who was going to properly do The God Emperor’s budding. Trouble in paradise?  Say it ain’t so!  Fake news!  Trust the phyzz!

Wrong, wrong, they’re always wrong.

Zman writes about the Neocons:

A sleazy dimwit like Goldberg accusing anyone of being mean spirited or stupid reveals a breathtaking lack of self-awareness. That is the very essence of the neoconservative, when you look at the shabby cast of characters flying the flag. Bill Kristol has been wrong about everything for decades, yet he shamelessly waddles around lecturing everyone, as if he is brilliant wise man. A normal man, possessing even a shred of decency, would be in hiding now if he had the record of Bill Kristol.

Rewritten:

A sleazy dimwit like [fill in the name of any “movement leader”] accusing anyone of being mean spirited or stupid reveals a breathtaking lack of self-awareness. That is the very essence of the Type I Quota Queen, when you look at the shabby cast of characters flying the flag.  “Movement leaders” have been wrong about everything for decades, yet they shamelessly waddle around lecturing everyone, as if they are brilliant wise men. A normal man, possessing even a shred of decency, would be in hiding now if he had the record of any of those losers.

Race, Brain Function, and Cortical Geometry

A remarkable hyper-confirmation of the biological basis of race.  In all cases, emphasis added.

Link to paper (including PDF).  Abstract, emphasis added:

Resting state functional magnetic resonance imaging (rs-fMRI) is a popular imaging modality for mapping the functional connectivity of the brain. Rs-fMRI is, just like other neuroimaging modalities, subject to a series of technical and subject level biases that change the inferred connectivity pattern. In this work we predicted genetic ancestry from rs-fMRI connectivity data at very high performance (area under the ROC curve of 0.93). Thereby, we demonstrated that genetic ancestry is encoded in the functional connectivity pattern of the brain at rest. Consequently, genetic ancestry constitutes a bias that should be accounted for in the analysis of rs-fMRI data.

This not only confirms a biological basis for race but does so in the most relevant and dramatic fashion possible: a scanning measurement of brain function is able to determine racial ancestry, correlated to genetics, in a highly accurate manner.  This is a “double win” for legitimate racial science: not only does it clearly demonstrate a biological basis for race, but shows that mental functioning at the physical (brain) level is correlated to genetically-determined racial identification, supporting hypotheses dealing with varying cognitive and behavioral mental characteristics associated with racial differences.

Also see this.

Here, we demonstrate that the three-dimensional geometry of cortical surface is highly predictive of individuals’ genetic ancestry in West Africa, Europe, East Asia, and America, even though their genetic background has been shaped by multiple waves of migratory and admixture events. The geometry of the cortical surface contains richer information about ancestry than the areal variability of the cortical surface, independent of total brain volumes. Besides explaining more ancestry variance than other brain imaging measurements, the 3D geometry of the cortical surface further characterizes distinct regional patterns in the folding and gyrification of the human brain associated with each ancestral lineage.

Intra-European differences:

BACKGROUND:
Human skull and brain morphology are strongly influenced by genetic factors, and skull size and shape vary worldwide. However, the relationship between specific brain morphology and genetically-determined ancestry is largely unknown.
METHODS:
We used two independent data sets to characterize variation in skull and brain morphology among individuals of European ancestry. The first data set is a historical sample of 1,170 male skulls with 37 shape measurements drawn from 27 European populations. The second data set includes 626 North American individuals of European ancestry participating in the Alzheimer’s Disease Neuroimaging Initiative (ADNI) with magnetic resonance imaging, height and weight, neurological diagnosis, and genome-wide single nucleotide polymorphism (SNP) data.
RESULTS:
We found that both skull and brain morphological variation exhibit a population-genetic fingerprint among individuals of European ancestry. This fingerprint shows a Northwest to Southeast gradient, is independent of body size, and involves frontotemporal cortical regions.
CONCLUSION:
Our findings are consistent with prior evidence for gene flow in Europe due to historical population movements and indicate that genetic background should be considered in studies seeking to identify genes involved in human cortical development and neuropsychiatric disease.

More excerpts:

Furthermore, Ashkenazi Jewish individuals are geographically dispersed in Europe and yet are genetically quite similar and genetically intermediate between SE European and Middle Eastern populations…

Indeed.

Intracranial and brain volumes and cortical surface area progressively increase with the amount of inferred NW European ancestry (fig. 3 b), and these measures are approximately 5% larger in the 10% of individuals with the most NW European ancestry compared to the 10% with the most SE European ancestry. This percentage increase matches the percentage increase in cranial length and breadth observed along the same NW-SE geographic axis in the skull data set (fig. 2 b) and cannot be attributed to a correlation with body size since we controlled for height and weight. This correlation involves specific – not global – brain morphology because hippocampal, basal ganglia, ventricular, and cerebellar volumes and average cortical thickness are not associated with NW-SE ancestry.

Next, we performed both a region of interest analysis and vertex-based tests across the cortex to test whether the surface area of specific cortical regions showed more significant association with the degree of NW-SE ancestry. We found that cortical surface area predominantly in the frontal and temporal lobes from both hemispheres is significantly associated (online suppl. table S4) and is 4–9% larger among 10% of individuals with the most NW European ancestry compared to 10% with the most SE European ancestry. We found a similar frontotemporal pattern of association with the degree of NW-SE ancestry with a vertex-based analysis (fig. 4 ; online suppl. fig. S6).

One can speculate that these differences are derived from Paleolithic hunter-gatherer vs. Neolithic farmer ancestry percentages, with brain size and intelligence being maximized among northern hunter-gatherers.  Note that the frontal lobes are important in judgment and impulse control, and these data may explain the stereotypes of the restrained, disciplined, stoic Nordic vs. the gesticulating, undisciplined, emotional Swarthoid. Temporal lobes deal with sensory input, and size differences there may be related to the greater requirement for spatial competence in hunter-gatherers.

Race Is Real.

Race Has A Genetic, Biological Basis.

Racial Differences Are Associated With Variation In Brain Activity and Brain Structure

The Good, the Bad, and the Ugly

A few examples of the spectrum of the “movement” and reactions to it.

First, the Good:

Codreanu speech.

Analysis of the Legionary movement.

Nice head gear on the groom.

Now, the Bad: the newest VDARE HBDer, the execrable fanboy Welton (emphasis added):

So, clearly, intelligence is the simplest explanation for most empirical evidence pf pathology.

Yes, and it is the simplest explanation for spelling errors as well.

Northeast Asians and Jews—even though they’re just as likely to be victims of racism…

Poor Asians and Jews tormented as they are by nasty racists in America.  Innocent, pure as freshly laid snow, these high-IQ supermen can save themselves from exposure to knuckle-dragging Euro-brutes by going back to Asia and Israel (in Asia as well, by the way), respectively.

…—do not display the same behavior patterns because they have high intelligence, meaning they do quite the opposite of what races with low intelligence do.

Never, never forget the fundamental basis of HBD: Jews and Asians have a high, high-IQ, and are your natural masters and betters.  All kneel before the Altar of Asia, and prostrate yourself before the Holy Yarmulke.

This model explains all the evidence. 

All, all, all!   And if, you know, reality contradicts HBD, we’ll just wave our hands around and try to explain away reality.

So it is amazing—considering that three of Benner’s five co-authors are Northeast Asians: Yishan Chen, Yijie Wang and Yen-Pi Cheng—that nobody thought of it.

Not so “amazing” when you consider that your Holy Orientals HATE White people, and will eagerly grasp on to, and promote, any theory that blames Whitey for Black and Hispanic deficiencies.  The Rising Tide of Color needs “high-IQ” leadership, and Jews and Asians are always ready and willing to oblige!

The issue of “depression” can be explained by the way that low IQ indirectly predicts depression, due to the fact that low IQ leads you into poverty, unemployment, relationship and friendship breakdown, drug abuse, alcoholism, poor health and thus general misery.

Sure!  Of course, HBD also tells us that Negroes have higher self-esteem, and lower suicide rates, than do Whites.  Don’t expect any consistency from HBDers – it’s a religious faith, a political movement, all dedicated to enslaving Whites to Jews and Asians.

By contrast, Northeast Asians are prone to depression for mainly genetic reasons and in spite of their tendency, in Western countries, to achieve high socioeconomic status. Evolved to extremely harsh yet predictable ecologies, their ancestors could only survive in tightly bonded groups. This meant that you had to be able to get on with everybody, or you’d be cast out into the cold to die. This selected in favor of strong social anxiety, which is adaptive because it means that you are concerned about the future. However, this can become overwhelming, leading to depression and suicide.

Can we ever imagine a more perfect example of a “just so” story?  Paging Rudyard Kipling!

Finally, the Ugly.

Look how communists and anarchists have degraded the beautiful Mota-Marin memorial, and consider modern official Spanish attitudes toward it. How about moving it to Romania?  You know, to all those deficient non-Westerners (according to the HBDers) dancing the hora (long may it turn)?

Race-mixers in Der Movement.

Another unbalanced Type I quota queen.

Diversity is a strength:

Behold the God Emperor

MAGA: Make Africans Great Again

It’s all in the Genes

Plain facts.  Excerpts presented. Emphasis added.

Establishment Lie:

Environment is everything; nurture (or lack of it) is the key.

Truth:

Now, one of the country’s top psychologists and behavioural geneticists, Professor Robert Plomin, of King’s College London, offers an emphatic conclusion.

It is drawn from 45 years of research and hundreds of studies. He says the single most important factor in each and every one of us — the very essence of our individuality — is our genetic make-up, our DNA.

The basic building blocks of life that we inherit from our parents are what determine who we are — not how much they loved us, read us books or which school they sent us to.

And this, by extension, must also apply to ethnic and racial differences.  All the societal manipulations in the world won’t make a Negro into a Dane.

DNA accounts for at least half the variance in people’s psychological traits, much more than any other single factor. Put simply, ‘nature’ trumps ‘nurture’ every time, and not just marginally, but by a long, long chalk.

Our DNA, fixed and unchangeable, determines whether we have a predisposition not just to physical traits — from how tall we are to how much we weigh — but also to our intelligence and our psychology, from a tendency to depression to having resilience and grit. 

Plomin’s revolutionary conclusion — outlined in a challenging and thought-provoking new book, Blueprint: How DNA Makes Us Who We Are — is a game-changer, he claims, with far-reaching implications for psychology and for society.

Chicago-born Plomin’s startling conclusions come from two of his long-term studies. Over the course of 40 years, he tracked 250 adopted children in Colorado along with the birth parents who gave them their genes, and the adoptive parents who raised them. After moving to London in 1994, he launched a 20-year study of more than 12,000 pairs of twins.

From these studies, it was possible to unravel the relative importance of genes as opposed to environment when it came to their development.

Millions of pieces of data were amassed from the parents, teachers and the children themselves, about psychological traits such as hyperactivity and inattention, talents such as school achievement and the ability to learn languages, and physical characteristics, such as the propensity to put on weight and become obese.

From all this, he found overwhelming evidence that adopted children are similar to their birth parents, not the parents who raised them. Identical twins (ie, from a single egg and therefore with the same DNA) develop much more similarly to each other as compared with non-identical twins (from separate eggs and with different DNA). 

The conclusion was clear — DNA makes us who we are. In the long term, the environment you grow up in has little impact on the way you turn out.

In fact, what really matters in such situations is our genes, because it is our genes that determine how well or badly an individual deals with such setbacks. And whether we’re resilient to life’s catastrophes or cave in is determined by our DNA, too.

Why shouldn’t the same apply to population groups?

In fact, Plomin argues, there are genetic influences in virtually everything we do. Those differences determine how we perceive and interpret the world we grow up in, and how we modify our behaviour accordingly.

As his research developed over the years, Plomin was taken by surprise by the all-pervasiveness of genetic influences he discovered in almost every aspect of human behaviour — even down to being a nice person or not.

Altruism, caring and kindness are components of what personality researchers call ‘agreeableness’, and for years it seemed logical to him that these traits had to be the result of the environment we live in and the influence of those around us.

But his research showed this was not the case. Being nice is also something in our DNA. The same goes for grit and determination. Nurture and example do not teach some children to be tougher than others, their genes do.

Consider differences between population groups in these traits.  Sorry, you cannot cherry pick and only apply these findings to atomized individuals.

All this leads Plomin to a conclusion that is hard to take: the family, he tells us, far from being the monolithic determinant of who we are, the bedrock from which we learn and grow, actually makes little difference to our personalities and the way we turn out.

The same applies to societies and races.

‘Each child is their own person genetically. We need to recognise and respect their genetic differences. If we go against the grain, we run the risk of damaging our relationship with them.’

Let’s rewrite:

Each race is their own population genetically. We need to recognise and respect their genetic differences. If we go against the grain, we run the risk of damaging our relationship with them – and risk damaging ourselves trying to fight Mother Nature.

Schools, he says, matter in that they teach basic skills such as literacy and numeracy. They also dispense fundamental information about history, science, maths and culture. But choice of school makes very little difference to a child’s achievement.

‘Genetics is by far the major source of individual differences in school achievement.’

As any multiracial society should be aware.

The same principle applies in the debate about private and state schools. If, as Plomin claims, schools have little effect on individual differences in achievement, then those 7 per cent of parents who pay huge sums to send their children to private schools in the belief that it will give them an advantage may well be wasting their cash.

Plomin writes: ‘Expensive schooling cannot survive a cost–benefit analysis on the basis of school achievement itself.’ 

If your genes fit, you’ll do well; and, if they don’t, no amount of cash can change the abilities you’re born with.

Negroes don’t perform badly because their schools are “bad.”  Their schools are bad because they’re the students in them.

Not that the influence of our DNA is confined to our early years when we’re growing up.

Indeed, Plomin shows that it gets stronger as we get older. More and more, we revert to type. Yes, other factors impact on us, such as our relationships with partners, children and friends, our jobs and interests. All contribute to give life meaning.

Which is why “Head Start” gains and other nonsense dissipate over time.

The same applies to anyone with a genetic propensity to depression, learning disabilities or alcohol abuse.

‘Genes are not destiny,’ says Plomin. You don’t have to succumb.

Perhaps, but you have limited options to change course, dependent upon your genetic blueprint.

It’s also good, he argues, that we can know our limits — those things that our DNA just won’t let happen, however hard we try.

Plomin quotes with approval the observation of American comedian W.C. Fields: ‘If at first you don’t succeed, try, try again. Then quit. There’s no use being a damn fool about it.’

Can we then give up attempting equal outcomes on the basis of race?

Plomin’s radical new world may force us to bow to our genetic limits but, on the plus side, it will encourage us, like Alastair Cook, to do the best we can with the talents we’ve been given.

Some folks can land on the moon, others can layup basketballs.  It’s all in the genes.

Three implications of this story:

1. The predictive value of gene pool to phenotype correlations will be greater for populations than for families, since the intra-family variability in phenotypic expression – due to the “meiotic lottery” – would tend to be averaged out over the millions of people making up typical ethnies.  Thus, one could more reliably predict phenotypic expression from the allele frequencies in the genepool when considering (large) populations.

2. Significant and long-term improvements in various psychometric performances would require genetic change – and the most rapid and directed approach to achieve this change (other than futuristic gene editing) would be via eugenics.

3. This all underscores the mendacity of globalist shills who tell us that people who lose their jobs due to free trade, outsourcing, immigration, and automation can simply be “educated” and “retrained” to perform the more challenging, information-based “jobs of the future.”  No, that middle-aged coal miner is not going to become the next Bill Gates or Elon Musk (and, besides, some of these “advanced jobs” will themselves be eventually lost to the same processes that have hollowed out the American [blue collar] working class and middle class).